Advertisement

AA amyloidosis is an emerging cause of nephropathy in obese patients

Published:February 14, 2017DOI:https://doi.org/10.1016/j.ejim.2017.02.004
      Dear Sir,

      Keywords

      To read this article in full you will need to make a payment

      Purchase one-time access:

      Academic & Personal: 24 hour online accessCorporate R&D Professionals: 24 hour online access
      One-time access price info
      • For academic or personal research use, select 'Academic and Personal'
      • For corporate R&D use, select 'Corporate R&D Professionals'

      Subscribe:

      Subscribe to European Journal of Internal Medicine
      Already a print subscriber? Claim online access
      Already an online subscriber? Sign in
      Institutional Access: Sign in to ScienceDirect

      References

        • Visser M.
        • Bouter L.M.
        • McQuillan G.M.
        • Wener M.H.
        • Harris T.B.
        Elevated C-reactive protein levels in overweight and obese adults.
        JAMA. 1999; 282: 2131-2135
        • Poitou C.
        • Viguerie N.
        • Cancello R.
        • De Matteis R.
        • Cinti S.
        • Stich V.
        • et al.
        Serum amyloid A: production by human white adipocyte and regulation by obesity and nutrition.
        Diabetologia. 2005; 48: 519-528
        • Alsina E.
        • Martin M.
        • Panadés M.
        • Fernández E.
        Renal AA amyloidosis secondary to morbid obesity?.
        Clin Nephrol. 2009; 72: 312-314
        • van der Heijden R.A.
        • Bijzet J.
        • Meijers W.C.
        • Yakala G.K.
        • Kleemann R.
        • Nguyen T.Q.
        • et al.
        Obesity-induced chronic inflammation in high fat diet challenged C57BL/6J mice is associated with acceleration of age-dependent renal amyloidosis.
        Sci Rep. 2015; 5: 16474
        • Adelman R.D.
        Obesity and renal disease.
        Curr Opin Nephrol Hypertens. 2002; 11: 331-335
        • Naeser P.
        • Westermark P.
        Amyloidosis in ageing obese-hyperglycemic mice and their lean litter-mates. A morphological study.
        Acta Pathol Microbiol Scand A. 1977; 85: 761-767
        • Cancello R.
        • Tordjman J.
        • Poitou C.
        • Guilhem G.
        • Bouillot J.L.
        • Hugol D.
        • et al.
        Increased infiltration of macrophages in omental adipose tissue is associated with marked hepatic lesions in morbid human obesity.
        Diabetes. 2006; 55: 1554-1561
        • Gómez-Ambrosi J.
        • Salvador J.
        • Rotellar F.
        • Silva C.
        • Catalán V.
        • Rodríguez A.
        • et al.
        Increased serum amyloid A concentrations in morbid obesity decrease after gastric bypass.
        Obes Surg. 2006; 16: 262-269
        • Lachmann H.J.
        • Goodman H.J.
        • Gilbertson J.A.
        • Gallimore J.R.
        • Sabin C.A.
        • Gillmore J.D.
        • et al.
        Natural history and outcome in systemic AA amyloidosis.
        N Engl J Med. 2007; 356: 2361-2371